10.1161/01.res.81.2.137
Crossref journal-article
Ovid Technologies (Wolters Kluwer Health)
Circulation Research (276)
Abstract

Abstract Programmed cell death (apoptosis) is recognized, increasingly, as a contributing cause of cardiac myocyte loss with ischemia/reperfusion injury, myocardial infarction, and long-standing heart failure. Although the exact mechanisms initiating apoptosis in these in vivo settings remain unproven, insights into the molecular circuitry controlling apoptosis more widely suggest the potential to protect mammalian ventricular muscle from apoptosis through one or more of these pathways, by pharmacological means or, conceivably, gene transfer.

Bibliography

MacLellan, W. R., & Schneider, M. D. (1997). Death by Design. Circulation Research, 81(2), 137–144.

Authors 2
  1. W. Robb MacLellan (first)
  2. Michael D. Schneider (additional)
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Dates
Type When
Created 13 years, 2 months ago (June 11, 2012, 8:47 p.m.)
Deposited 1 year, 3 months ago (May 12, 2024, 4:44 p.m.)
Indexed 1 month, 2 weeks ago (July 14, 2025, 11:47 p.m.)
Issued 28 years ago (Aug. 1, 1997)
Published 28 years ago (Aug. 1, 1997)
Published Print 28 years ago (Aug. 1, 1997)
Funders 0

None

@article{MacLellan_1997, title={Death by Design: Programmed Cell Death in Cardiovascular Biology and Disease}, volume={81}, ISSN={1524-4571}, url={http://dx.doi.org/10.1161/01.res.81.2.137}, DOI={10.1161/01.res.81.2.137}, number={2}, journal={Circulation Research}, publisher={Ovid Technologies (Wolters Kluwer Health)}, author={MacLellan, W. Robb and Schneider, Michael D.}, year={1997}, month=aug, pages={137–144} }