DOI: 10.1055/s-0038-1653178. Acquired α2 Antiplasmin Deficiency In Patients With Glomerular Proteinuria

Acquired α2 Antiplasmin Deficiency In Patients With Glomerular Proteinuria

10.1055/s-0038-1653178

Crossref proceedings-article

Schattauer GmbH

Thrombosis and Haemostasis (194)

Abstract

α2 antiplasmin has been measured in the plasma of 20 patients with a glomerularproteinuria. Significantly lower levels were found as compared to eight controls. The unconcentrated urine was also examined and α2 antiplasmin antigen was demonstrated in seven patients but in none of the controls. Deficiency of antithrombin III due to glomerular leak has been suggested as a major cause for tendency for venous thrombosis in these patients.Acquired α2 antiplasmin deficiency also due to renal loss may tend to counterbalance this effect. Furthermore, the presence of this potent plasmin inhibitor in the urine in patients with nephrotic syndrome may be involved in other pathological mechanisms involving fibrinolysis in the renal tract.

Bibliography

Taberner, D. A. (1981). Acquired Î±2 Antiplasmin Deficiency In Patients With Glomerular Proteinuria. Oral Presentations.

Authors 1

D A Taberner (additional)

References 0 Referenced 1

None

Dates

Type	When
Created	7 years, 1 month ago (July 25, 2018, 2:43 p.m.)
Deposited	5 years, 8 months ago (Dec. 6, 2019, 12:16 p.m.)
Indexed	3 years, 5 months ago (April 3, 2022, 12:47 a.m.)
Issued	44 years, 8 months ago (Jan. 1, 1981)
Published	44 years, 8 months ago (Jan. 1, 1981)
Published Online	7 years, 1 month ago (July 25, 2018)
Published Print	44 years, 8 months ago (Jan. 1, 1981)

Funders 0

None

BibTeX

@inproceedings{Taberner_1981, title={Acquired α2 Antiplasmin Deficiency In Patients With Glomerular Proteinuria}, ISSN={2567-689X}, url={http://dx.doi.org/10.1055/s-0038-1653178}, DOI={10.1055/s-0038-1653178}, booktitle={Oral Presentations}, publisher={Schattauer GmbH}, author={Taberner, D A}, year={1981} }

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