Crossref
journal-article
Elsevier BV
Cell (78)
Authors
11
- Akimitsu Konishi (first)
- Shigeomi Shimizu (additional)
- Junko Hirota (additional)
- Toshifumi Takao (additional)
- Yuhong Fan (additional)
- Yosuke Matsuoka (additional)
- Lilin Zhang (additional)
- Yoshihiro Yoneda (additional)
- Yoshitaka Fujii (additional)
- Arthur I. Skoultchi (additional)
- Yoshihide Tsujimoto (additional)
References
59
Referenced
267
10.1126/science.281.5381.1322
/ Science / The Bcl-2 protein family by Adams (1998)10.1007/s004390050630
/ Hum. Genet. / The human histone gene cluster at the D6S105 locus by Albig (1997)10.1042/bj3450271
/ Biochem. J. / Bax oligomerization is required for channel-forming activity in liposomes and to trigger cytochrome c release from mitochondria by Antonsson (2000)10.1074/jbc.M010810200
/ J. Biol. Chem. / Bax is present as a high molecular weight oligomer/complex in the mitochondrial membrane of apoptotic cells by Antonsson (2001)10.1101/gad.14.6.704
/ Genes Dev. / PERP, an apoptosis-associated target of p53, is a novel member of the PMP-22/gas3 family by Attardi (2000)10.1007/s004120050382
/ Chromosoma / Nucleo-cytoplasmic translocation of histone H1 during the HeLa cell cycle by Bleher (1999)10.1016/S0092-8674(00)81732-8
/ Cell / Apaf1 (CED-4 homolog) regulates programmed cell death in mammalian development by Cecconi (1998)10.1074/jbc.272.13.8113
/ J. Biol. Chem. / Histone H1b phosphorylation is dependent upon ongoing transcription and replication in normal and ras-transformed mouse fibroblasts by Chadee (1997)10.1038/362849a0
/ Nature / Thymocyte apoptosis induced by p53-dependent and independent pathways by Clarke (1993)10.1083/jcb.144.5.891
/ J. Cell Biol. / Bid-induced conformational change of Bax is responsible for mitochondrial cytochrome c release during apoptosis by Desagher (1999)10.1038/12469
/ Nat. Med. / Anti-cancer activity of targeted pro-apoptotic peptides by Ellerby (1999)10.1074/jbc.M202396200
/ J. Biol. Chem. / The apoptotic protein tBid promotes leakage by altering membrane curvature by Epand (2002)10.1128/MCB.21.21.7268-7276.2001
/ Mol. Cell. Biol. / Bid, a widely expressed proapoptotic protein of the Bcl-2 family, displays lipid transfer activity by Esposti (2001)10.1128/MCB.21.23.7933-7943.2001
/ Mol. Cell. Biol. / Individual somatic H1 subtypes are dispensable for mouse development even in mice lacking the H1(0) replacement subtype by Fan (2001)10.1038/416560a
/ Nature / p63 and p73 are required for p53-dependent apoptosis in response to DNA damage by Flores (2002)10.1016/S0167-4781(98)00062-1
/ Biochim. Biophys. Acta / Expression of murine H1 histone genes during postnatal development by Franke (1998)10.1038/36894
/ Nature / CRM1 is responsible for intracellular transport mediated by the nuclear export signal by Fukuda (1997)10.1083/jcb.144.5.903
/ J. Cell Biol. / Cell damage-induced conformational changes of the pro-apoptotic protein Bak in vivo precede the onset of apoptosis by Griffiths (1999)10.1093/emboj/17.14.3878
/ EMBO J. / Enforced dimerization of BAX results in its translocation, mitochondrial dysfunction and apoptosis by Gross (1998)10.1016/S1097-2765(01)00258-1
/ Mol. Cell / The Peutz-Jegher gene product LKB1 is a mediator of p53-dependent cell death by Karuman (2001)10.1126/science.270.5233.96
/ Science / Bax-deficient mice with lymphoid hyperplasia and male germ cell death by Knudson (1995)10.1042/bj3370319
/ Biochem. J. / Changes in the protein pattern of H1 histones associated with apoptotic DNA fragmentation by Kratzmeier (1999)10.1016/S0021-9258(18)33251-4
/ J. Biol. Chem. / The histone H1 complements of dividing and nondividing cells of the mouse by Lennox (1983)10.1126/science.289.5482.1159
/ Science / Cytochrome c release and apoptosis induced by mitochondrial targeting of nuclear orphan receptor TR3 by Li (2000)10.1016/S1097-2765(00)00136-2
/ Mol. Cell / The combined functions of proapoptotic Bcl-2 family members Bak and Bax are essential for normal development of multiple tissues by Lindsten (2000)10.1038/362847a0
/ Nature / p53 is required for radiation-induced apoptosis in mouse thymocytes by Lowe (1993)10.1016/0092-8674(93)90719-7
/ Cell / p53-dependent apoptosis modulates the cytotoxicity of anticancer agents by Lowe (1993)10.1074/jbc.C000048200
/ J. Biol. Chem. / Mitochondrial translocation of protein kinase C δ in phorbol ester-induced cytochrome c release and apoptosis by Majumder (2000)10.1074/jbc.275.21.16202
/ J. Biol. Chem. / Death signal-induced localization of p53 protein to mitochondria. A potential role in apoptotic signaling by Marchenko (2000)10.1242/jcs.107.3.693
/ J. Cell Sci. / Exogenous histone H1 injection into mitotic cells disrupts synchronous progression of mitotic events by delaying chromosome decondensation by Matsuoka (1994)10.1016/0092-8674(95)90412-3
/ Cell / Tumor suppressor p53 is a direct transcriptional activator of the human bax gene by Miyashita (1995)10.1016/S1097-2765(01)00214-3
/ Mol. Cell / PUMA, a novel proapoptotic gene, is induced by p53 by Nakano (2001)10.1126/science.288.5468.1053
/ Science / Noxa, a BH3-only member of the Bcl-2 family and candidate mediator of p53-induced apoptosis by Oda (2000)10.1016/S0092-8674(00)00073-8
/ Cell / p53AIP1, a potential mediator of p53-dependent apoptosis, and its regulation by Ser-46-phosphorylated p53 by Oda (2000)10.1016/0167-4838(90)90116-W
/ Biochim. Biophys. Acta / The histone H1 globular region. A possible supersecondary structure from spectroscopic and statistical studies by Pepe (1990)10.1038/38525
/ Nature / A model for p53-induced apoptosis by Polyak (1997)10.1038/sj.onc.1203150
/ Oncogene / Damage-induced apoptosis in intestinal epithelia from bcl-2-null and bax-null mice by Pritchard (1999)10.1038/sj.cdd.4400998
/ Cell Death Differ. / Keeping killers on a tight leash by Puthalakath (2002)10.1083/jcb.146.5.905
/ J. Cell Biol. / Megabase chromatin domains involved in DNA double-strand breaks in vivo by Rogakou (1999)10.1126/science.274.5290.1194
/ Science / Ultraviolet light and osmotic stress by Rosette (1996){'key': '10.1016/S0092-8674(03)00719-0_BIB41', 'first-page': '2251', 'article-title': 'Bcl-2 expression prevents activation of the ICE protease cascade', 'volume': '12', 'author': 'Shimizu', 'year': '1996', 'journal-title': 'Oncogene'}
/ Oncogene / Bcl-2 expression prevents activation of the ICE protease cascade by Shimizu (1996)10.1073/pnas.95.4.1455
/ Proc. Natl. Acad. Sci. USA / Bcl-2 prevents apoptotic mitochondrial dysfunction by regulating proton flux by Shimizu (1998)10.1242/dev.128.5.655
/ Development / A mammalian oocyte-specific linker histone gene H1oo by Tanaka (2001)10.1038/382816a0
/ Nature / Cooperation of the tumour suppressors IRF-1 and p53 in response to DNA damage by Tanaka (1996)10.1002/jcp.10254
/ J. Cell. Physiol. / Cell death regulation by the Bcl-2 protein family in the mitochondria by Tsujimoto (2003)10.1016/S0014-5793(99)01761-5
/ FEBS Lett. / Bcl-2 family by Tsujimoto (2000)10.1038/4133
/ Nat. Struct. Biol. / Location and function of linker histones by Vignali (1998)10.1110/ps.9.4.627
/ Protein Sci. / A helix-turn motif in the C-terminal domain of histone H1 by Vila (2000){'key': '10.1016/S0092-8674(03)00719-0_BIB49', 'first-page': '2922', 'article-title': 'The expanding role of mitochondria in apoptosis', 'volume': '15', 'author': 'Wang', 'year': '2001', 'journal-title': 'Genes Dev.'}
/ Genes Dev. / The expanding role of mitochondria in apoptosis by Wang (2001)10.1006/jmbi.1997.1166
/ J. Mol. Biol. / The mouse histone H1 genes by Wang (1997)10.1101/gad.14.16.2060
/ Genes Dev. / tBID, a membrane-targeted death ligand, oligomerizes BAK to release cytochrome c by Wei (2000)10.1126/science.1059108
/ Science / Proapoptotic BAX and BAK by Wei (2001)10.1038/ng0897-397
/ Nat. Genet. / ATM and p53 cooperate in apoptosis and suppression of tumorigenesis, but not in resistance to acute radiation toxicity by Westphal (1997)10.1016/S0960-9822(02)00736-4
/ Curr. Biol. / Apoptosis by Wolf (2002)10.1083/jcb.139.5.1281
/ J. Cell Biol. / Movement of Bax from the cytosol to mitochondria during apoptosis by Wolter (1997)10.1074/jbc.M207516200
/ J. Biol. Chem. / Bcl-xL protects BimEL-induced Bax conformational change and cytochrome c release independent of interacting with Bax or BimEL by Yamaguchi (2002)10.1016/S0092-8674(00)81733-X
/ Cell / Apaf1 is required for mitochondrial pathways of apoptosis and brain development by Yoshida (1998)10.1016/S1097-2765(01)00213-1
/ Mol. Cell / PUMA induces the rapid apoptosis of colorectal cancer cells by Yu (2001)10.1016/0304-4157(95)00003-A
/ Biochim. Biophys. Acta / The mitochondrial permeability transition by Zoratti (1995)
Dates
Type | When |
---|---|
Created | 21 years, 4 months ago (April 17, 2004, 2:47 p.m.) |
Deposited | 3 years, 2 months ago (June 25, 2022, 12:35 p.m.) |
Indexed | 6 days, 5 hours ago (Aug. 26, 2025, 2:29 a.m.) |
Issued | 22 years ago (Sept. 1, 2003) |
Published | 22 years ago (Sept. 1, 2003) |
Published Print | 22 years ago (Sept. 1, 2003) |
Funders
2
Ministry of Education, Culture, Sports, Science and Technology
10.13039/501100001700
Region: Asia
gov (National government)
Labels
3
- Monbu-kagaku-shō
- 文部科学省
- MEXT
Japan Science and Technology Agency
10.13039/501100002241
Region: Asia
gov (National government)
Labels
7
- SCIENCE AND TECHNOLOGY AGENCY OF JAPAN
- 国立研究開発法人科学技術振興機構
- 国立研究開発法人科学技術振興機構 japan science and technology agency
- Japan Science and Technology Agency (JST)
- かがくぎじゅつしんこうきこう
- 科学技術振興機構
- JST
@article{Konishi_2003, title={Involvement of Histone H1.2 in Apoptosis Induced by DNA Double-Strand Breaks}, volume={114}, ISSN={0092-8674}, url={http://dx.doi.org/10.1016/s0092-8674(03)00719-0}, DOI={10.1016/s0092-8674(03)00719-0}, number={6}, journal={Cell}, publisher={Elsevier BV}, author={Konishi, Akimitsu and Shimizu, Shigeomi and Hirota, Junko and Takao, Toshifumi and Fan, Yuhong and Matsuoka, Yosuke and Zhang, Lilin and Yoneda, Yoshihiro and Fujii, Yoshitaka and Skoultchi, Arthur I. and Tsujimoto, Yoshihide}, year={2003}, month=sep, pages={673–688} }